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( Yeast Infection)

by Stephen A. Levine, Ph. D & Larry Jordan


Candida Albicans is a normal fungus inhabiting the mouth, throat and gastrointestinal tract. Excess development may result from certain types of life style and can be an indicator of immune status. Scientifically classified as fungus, these yeast organisms are free of chlorophyll and therefore cannot by photosynthesis make their own food from sunlight; hence they depend on their host for nourishment. They use oxygen for metabolism.

Candida can be cultured from the mouth, vagina, or feces from most people, and more frequently from debilitated individuals. Yeasts such as candida are often secondary invaders in other infections. Traditional beliefs hold that those most susceptible to candida infections are infants (thrush), women (vaginitis), persons who have undergone extended antibiotic therapy or used birth control pills, and postoperative patients. (1)


  • The use of Antibiotics
  • Birth Control Pills
  • Cortisone
  • Sugar & fat in the diet
  • Yeast in the diet
  • Insufficient supply of nutrients
  • A genetic deficiency of Ig A (Immunoglobulin A)
  • Drugs or stress induced immune depression and related diseases
  • Hypothyroidism - this is a condition that is fairly common, as a low body temperature
  • permits easier and faster growth of candida albicans


In the last five years much interest has developed with the recognition that candida overgrowth can underlie common gastrointestinal and genitourinary tract conditions such as:

  • Indigestion
  • Heartburn
  • Bloating
  • Cystitis
  • Anal itching
  • Vaginitis
  • AsthmaHives
  • Acne
  • Hay fever
  • Bronchitis
  • Earaches
  • Mental & Emotional problems including:
  • Headache
  • Confusion
  • Extreme irritability
  • Depression
  • Memory lapses
  • Lethargy

Proliferation of Candida will cause damage to the gastrointestinal mucosa and eventually allow absorption of the organism into the blood stream so that it will reach other tissues. Other common yeast associated problems are related to malfunction of glands and other organs, such as hypothyroid or hyperthyroid conditions, adrenal failure, ovarian failure, hormone deficiencies, etc. (2)


  • Oxygen-carrying red blood cells are rigid and stiff when compared to patients without candida
  • Fatigue
  • Carbohydrate craving
  • Topical infections of fingernails, vaginal infections, underarm infections
  • White coated tongue
  • Increased allergic symptoms
  • Reduced thyroid function
  • Lowered immune system and increased susceptibility to viral infections
  • T-cell helper/suppressor ratios radically lowered
  • Staphylococcus danger increases as much as 1000,000 times
  • Up to 3/4 of the lymphocytes become paralyzed
  • It produces the chemical toxin acetaldehyde - the precursor of ethyl alcohol and the first by-product of alcohol's oxidation. This toxin depresses the citric acid cycle (by which the body produces energy from food) and disrupts collagen production and fatty acid oxidation. It blocks synapses - the junctions between neurons or between a neuron and an organ, where electrical or chemical impulses are transmitted. It also binds to neurotransmitters and causes brain symptoms. Acetaldehyde is a close molecular cousin to formaldehyde, and the two toxic substances are metabolized similarly by the body. Thus exposure to formadlehyde in carpeting, furniture or particleboard often causes candidiasis to worsen.
  • Candida seems to upset the function of organs as opposed to damaging them.


  • Allergic symptoms
  • Asthma
  • Skin rashes
  • Hormonal blockages
  • Learning disabilities
  • Hyperactivity
  • Multiple sclerosis
  • Spots in the eyes
  • Lupus
  • Crohn's disease
  • Psychiatric problems
  • Ear infections
  • Fungus infections
  • Vaginitis
  • Rheumatoid arthritis
  • Staph infections
  • Toxic shock syndrome
  • Hypersensitivity to chemicals and foods



  1. Krupp and Chatton: Current Medical Diagnosis and Treatment; 1982, Lange.
  2. Truss, Orien: The Missing Diagnosis.
  3. Levine, S. and Kidd, Parris: Antioxidant Adaptation: Its Role in Free Radical Biochemistry; 1985, Biocurrents Press, San Francisco. Spallnolz, T.: Selenium in Biology and Medicine; Aui Publishing Co., Inc., 1981.
  4. Wuepper, Ray: Investiga. Dermatol. 67 7003, 1976
  5. Truss, Orien: Journal of Orthomolecular Psychiatry, Vol. 2, 1984.
  6. Rodex, G.E., et al.: New Zealand Medical Journal, August 10, 1983.
  7. Levine, S. and Kidd, Parris: Beyond Antioxidant Adaptation, Journal of Orthomolecular Psychiatry, 1985, Vol. 14, No. 3.
  8. Levine, S. and Kidd, P.: Manuscript in preparation, 1985, San Francisco.
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